Cancer of the Liver, Esophagus, and Nasopharynx by Zhang You-Hui (auth.), Professor Dr. med. Gustav Wagner,

By Zhang You-Hui (auth.), Professor Dr. med. Gustav Wagner, Professor Zhang You-Hui (eds.)

To stimulate larger German-Chinese cooperation within the box of oncology, the Ger­ guy melanoma learn middle (DKFZ) carried via a "Chinese-German Sympo­ sium on melanoma of the Liver, the Esophagus, and the Nasopharynx" in Heidelberg from three to five July 1985. those 3 kinds of melanoma have been selected as they're tremendous infrequent in Europe yet one of the most usually came across types in China. virtually half the approximately 260000 situations of fundamental liver melanoma happening each year world wide are chinese language; of the approximately 300000 esophagus cancers taking place every year, greater than part are chinese language; nasopharyngeal melanoma is approximately a hundred instances extra common between chinese language humans than in Caucasians. less than those conditions, it isn't awesome that the chinese language visitor audio system of the Heidelberg symposium - chosen experts within the 3 subsections of oncolo­ gy pointed out - have been capable of file approximately wide reviews with sufferer popu­ lations of a dimension infrequently conceivable in Europe. the nice medical adventure of the chinese language visitors was once supplemented by way of unique contributions of eu specialists. hence, this quantity allows a topical survey of the state-of-the-art in those 3 can­ cers, with subsections of epidemiology, pathology, and medical therapy. The editors desire to thank the Federal Ministry of analysis and know-how (BMFT) for his or her encouragement and direct assist in getting ready and wearing during the symposium, the administration Board of the DKFZ for his or her beneficiant counsel and Springer-Verlag for the e-book and ideal creation of this volume.

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Cancer of the Liver, Esophagus, and Nasopharynx Edited by G. ".... ..... '. •• "' .... ,'to'. •. ' {' .. " ,-. : . , . • ,0 ~"o • : ••• ,': ' ' . ' . ": ... :; .. : " .. . - .. - ' .. ', I'. : ° 0 ,', :. • .... • Fig. 1. Autoradiogram of regenerating rat liver 60 min after [3H]-thymidine injection at 43 h after partial hepatectomy and 4 h after a continuous intravenous infusion of HU from 14 to 39 h after partial hepatectomy. About 80% of the hepatocytes are simultaneously in DNA synthesis intralobular localization of hepatocytes [18], with periportally located liver cells being the first to enter S phase and the perivenous hepatocytes starting DNA synthesis after a delayed 0 1 phase.

Taking the frequency of ATPase-deficient foci in the liver as a marker for the initiating action of a carcinogen, it is possible to determine the initiation sensitivity of hepatocytes during specific stages of proliferation after partial hepatectomy. Investigations in the past have shown that partial resection of the liver fails to induce a synchronous start of DNA synthesis in hepatocytes of the remaining liver. The Gj/S transit is scattered from 15 to 40 h after the operation, depending on the * The original work reported in this paper was supported by grants from the Deutsche Forschungsgemeinschaft, Bonn, and Dr.

Liver tumors could be detected by scintigrams, ultrasonograms, biopsies, or autopsies in many patients with this genetically fixed disease. Whereas the tumors were relatively rare in the 1st decade oflife, they appeared in all patients who survived adolescence. Histologically, both adenomas and carcinomas were diagnosed, and in some cases the clinical course of the disease suggested transformation from adenomas into carcinomas. The observations in humans lend further support to the hypothesis that the metabolic disturbance leading to the hepatocellular glycogenosis, which is probably fixed at the genetic level in both humans and experimental animals, might also be causally related to the neoplastic transformation of the hepatocytes [5, 11].

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